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The molecular mechanisms proposed to explain how ROS could specifically mediate brain damage are the following: (1) The lower levels of antioxidants can promote increased activity of lipoxygenase in CNS stimulating leukotriene production, thereby increasing the immunoinflammatory processes in the cerebral cortex

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The NF-κB pathway is a complex signaling cascade that regulates the expression of various genes involved in immunity, inflammation, and cell survival (96)