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doi: 10.1172/JCI4165 32 LeeSCHanSHLiJJLeeSHJungD-SKwakS-Jet al

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What the published work does support is cellular, and it is the subject of the sections below: GHK-Cu shifts fibroblast gene expression, raises decorin (which organises collagen fibrils), increases type I and III collagen without raising TGF-β, limits the persistence of myofibroblasts — the cells behind raised, contracted scars — and restores growth-factor output in irradiated fibroblasts