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Notably, while the prevalence of DPN is considered to be rather low within the first five years of disease onset in T1D, slowing of nerve conduction velocity may be one of the earliest neuropathic abnormalities in T2D, often present at diagnosis (23, 24)

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The interaction between α-synuclein and microglial TLR2 promotes the growth and spread of α-synuclein pathology [69]